Part:BBa_K3866014
TANGO module NOT GATE SCFAs detection
Usage and Biology
This composite part constitutes the G-protein coupled bioreceptor composite of the dual TANGO-GPCR assay(Dogra, Sona, Kumar and Yadav, 2016) (the other part is the b-arrestin-2 constituent, placed under the control of the arabinosed-induced promoter. FFAR2 is a naturally found eukaryotic GPCR protein that exhibits high affinity for SCFAs (acetic, propionic and butyric acid)(Kaemmerer, 2010) . After background searching through the NCBI database, we have identified the gene coding sequences needed for the designing of this gene construct. More specifically, a vasopressin receptor 2 segment has been attached to the C-terminal of the receptor, as it mediates recruitment of a TEV tagged b-arrestin-2, along with a TEV cleavage site. When TEV protease cleaves , the lac repressor is released and binds to the lac operator . In the presence of SCFAs the GPCR is activated.
Design Notes
The coding sequence was domesticated . We removed BsmBI ,BsaI , BtgZI, BpiI sites in order to be compatible with GoldenBraid and MoClo. The sequence is cloned in seva alpha1R vector BBa_K3505008and has overhangs compatible for GoldenBraid cloning.
Verification of Cloning
Exerimental Use and Experience
This TANGO NOT GATE after SCFAs induction should lower the Fluorecence.
Cultures grown in LB and induced with 1% L-arabinose for 3 hours. t=0 is considered when SCFAs are added.
- The positive control is the TU with ECFP only BBa_K3505032 (LacO)
- The negative controls are the empty vector BBa_K3505008with and without SCFAs.(a1R)
- The lines in between are TANGO modules with 0, 0,1, 1 and 10 mM Sodium Acetate(GPCR)
Conclusion
We observe that there was continuous non-specific suppression, both in the presence and absence of SCFAs. We hypothesize that 1. either suppression takes place without the release of the LacI from the GPCR before it reaches the membrane, or 2. the TEV protease cleaves the LacI non-specifically.
Sequence and Features
- 10INCOMPATIBLE WITH RFC[10]Illegal SpeI site found at 8313
Illegal PstI site found at 3407
Illegal PstI site found at 3987
Illegal PstI site found at 7005
Illegal PstI site found at 7092 - 12INCOMPATIBLE WITH RFC[12]Illegal NheI site found at 8817
Illegal NheI site found at 8840
Illegal SpeI site found at 8313
Illegal PstI site found at 3407
Illegal PstI site found at 3987
Illegal PstI site found at 7005
Illegal PstI site found at 7092 - 21INCOMPATIBLE WITH RFC[21]Illegal BglII site found at 6905
Illegal BamHI site found at 1148
Illegal BamHI site found at 3028
Illegal BamHI site found at 3229
Illegal BamHI site found at 6607
Illegal XhoI site found at 7302
Illegal XhoI site found at 7696 - 23INCOMPATIBLE WITH RFC[23]Illegal SpeI site found at 8313
Illegal PstI site found at 3407
Illegal PstI site found at 3987
Illegal PstI site found at 7005
Illegal PstI site found at 7092 - 25INCOMPATIBLE WITH RFC[25]Illegal SpeI site found at 8313
Illegal PstI site found at 3407
Illegal PstI site found at 3987
Illegal PstI site found at 7005
Illegal PstI site found at 7092
Illegal NgoMIV site found at 3487
Illegal AgeI site found at 983
Illegal AgeI site found at 2863
Illegal AgeI site found at 6442
Illegal AgeI site found at 7288 - 1000INCOMPATIBLE WITH RFC[1000]Illegal SapI site found at 965
Illegal SapI site found at 2845
Illegal SapI site found at 3683
Illegal SapI site found at 3938
Illegal SapI site found at 6424
Illegal SapI.rc site found at 3140
Illegal SapI.rc site found at 8247
Illegal SapI.rc site found at 8595
References
- Dogra, S., Sona, C., Kumar, A. and Yadav, P., 2016. Tango assay for ligand-induced GPCR–β-arrestin2 interaction. Methods in Cell Biology, pp.233-254.
- Kaemmerer, E., 2010. Fatty acid binding receptors in intestinal physiology and pathophysiology. World Journal of Gastrointestinal Pathophysiology, 1(5), p.147.
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