Difference between revisions of "Part:BBa K1202107"

 
 
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__NOTOC__
 
__NOTOC__
 
<partinfo>BBa_K1202107 short</partinfo>
 
<partinfo>BBa_K1202107 short</partinfo>
  
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This part has two component.
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The general attitude to our parts is to designate alternative parts. For cancer killing mechanism, we searched through literature and found E4orf4 peptide. This derivative prospective cancer drug, like apoptin, induces apoptosis in cancer cells, selectively. Moreover, in literature and, of course iGEM, TAT-E4orf4 combination is completely novel.
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Tat: Tat protein is entering into this complex to the cell owing to dissolving cell membrane.
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E4ORF4: E4ORF4 is killing cancer cell owing to forcing cancer cells to apoptosis.
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<!-- Add more about the biology of this part here
 
<!-- Add more about the biology of this part here

Latest revision as of 00:45, 5 October 2013

TAT-E4-orf4: Cancer killing peptide

The general attitude to our parts is to designate alternative parts. For cancer killing mechanism, we searched through literature and found E4orf4 peptide. This derivative prospective cancer drug, like apoptin, induces apoptosis in cancer cells, selectively. Moreover, in literature and, of course iGEM, TAT-E4orf4 combination is completely novel.

Sequence and Features


Assembly Compatibility:
  • 10
    COMPATIBLE WITH RFC[10]
  • 12
    INCOMPATIBLE WITH RFC[12]
    Illegal NheI site found at 7
    Illegal NheI site found at 30
  • 21
    COMPATIBLE WITH RFC[21]
  • 23
    COMPATIBLE WITH RFC[23]
  • 25
    INCOMPATIBLE WITH RFC[25]
    Illegal NgoMIV site found at 158
    Illegal NgoMIV site found at 167
  • 1000
    COMPATIBLE WITH RFC[1000]