Difference between revisions of "Part:BBa K2558211:Design"
Line 1: | Line 1: | ||
+ | __NOTOC__ | ||
+ | <partinfo>BBa_K2558211 short</partinfo> | ||
+ | <partinfo>BBa_K2558211 SequenceAndFeatures</partinfo> | ||
+ | |||
+ | |||
+ | ===Design Notes=== | ||
+ | With the hope to find an optimal promotor for our NEON system, we designed 9 mutations on sites 18, 19 and 20 near the luxR binding site and tested one on -10 site that TUST 2017 concluded to show decreased leakage. We conducted experiments to evaluate these lux pR mutants’ reaction to AHL stimulation and their leakage level. The test devices we designed include a constantly expressed luxR and a lux pR (or mutant) (like BBa_K2558211 with original lux pR promotor https://parts.igem.org/Part:BBa_K2558211, and BBa_K2558212 with lux pR-HS promotor https://parts.igem.org/Part:BBa_K2558212). | ||
+ | |||
+ | |||
+ | ===Source=== | ||
+ | |||
+ | |||
+ | ===References=== | ||
+ | Koch B et al. The LuxR receptor: the sites of interaction with quorum-sensing signals and inhibitors. Microbiology. 2005 Nov;151(Pt 11):3589-602. |
Revision as of 06:34, 15 October 2018
lux pR test device
- 10COMPATIBLE WITH RFC[10]
- 12INCOMPATIBLE WITH RFC[12]Illegal NheI site found at 1074
Illegal NheI site found at 1097 - 21COMPATIBLE WITH RFC[21]
- 23COMPATIBLE WITH RFC[23]
- 25COMPATIBLE WITH RFC[25]
- 1000INCOMPATIBLE WITH RFC[1000]Illegal SapI.rc site found at 177
Design Notes
With the hope to find an optimal promotor for our NEON system, we designed 9 mutations on sites 18, 19 and 20 near the luxR binding site and tested one on -10 site that TUST 2017 concluded to show decreased leakage. We conducted experiments to evaluate these lux pR mutants’ reaction to AHL stimulation and their leakage level. The test devices we designed include a constantly expressed luxR and a lux pR (or mutant) (like BBa_K2558211 with original lux pR promotor https://parts.igem.org/Part:BBa_K2558211, and BBa_K2558212 with lux pR-HS promotor https://parts.igem.org/Part:BBa_K2558212).
Source
References
Koch B et al. The LuxR receptor: the sites of interaction with quorum-sensing signals and inhibitors. Microbiology. 2005 Nov;151(Pt 11):3589-602.