Difference between revisions of "Part:BBa K1456015:Experience"
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| + | <ul>(p<0,05 and p<0,01 for H2O2 and TNFα induced kappaB-luc,respectively.)<br/> | ||
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<li>At the end of the experiment, we could not see an increase in the luciferase assay of cells exposed to H202. We assumed that cell death due to the direct exposal of H2O2 was what limited our luciferase activity expectations. Our assumptions were further supported after the microscope analysis we performed, which showed that our cells were unattached from the plates upon being exposed to H202. | <li>At the end of the experiment, we could not see an increase in the luciferase assay of cells exposed to H202. We assumed that cell death due to the direct exposal of H2O2 was what limited our luciferase activity expectations. Our assumptions were further supported after the microscope analysis we performed, which showed that our cells were unattached from the plates upon being exposed to H202. | ||
Latest revision as of 14:54, 21 October 2014
- (p<0,05 and p<0,01 for H2O2 and TNFα induced kappaB-luc,respectively.)
- At the end of the experiment, we could not see an increase in the luciferase assay of cells exposed to H202. We assumed that cell death due to the direct exposal of H2O2 was what limited our luciferase activity expectations. Our assumptions were further supported after the microscope analysis we performed, which showed that our cells were unattached from the plates upon being exposed to H202.
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